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So the virus enters the epithelial cell and the
Cell Physics causes the RNA to be discharged into the cell medium,
starting the self-replication Biosynthesis until the structural
integrity of the cell is breached, and the replicated viral RNAs are
discharged into the inter-cell spaces causing secondary infection
spreading in the body.
Of research interest is the form of the virus
discharged from the primary infections: The viral RNA as replicated, The
pre-infection virus-morphology, as is implicitly assumed. Discovering
the discharge form of the virus determines the mechanism of the
infection spread, and hence infection-mechanism changes that define
Infection Evolution Dynamics and help developing effective drugs and
vaccines, as well as determine which vaccine should be efficacious,
given that vaccine development is on two thrusts of biologics adopting
for stimulating immune response: The Spike-trimer biologic, A viral-RNA
biologic.
Rationally, replicated viral RNAs should not
re-encase into the pre-infection morphology, as continued prevailing of
the Cell Physics that caused the discharge of the initial infecting RNA.
Inferentially, the external and internal infection mechanisms are
different. |
